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Coloff (Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA) for helpful commentary and discussion

This article compares the two head-to-head on mechanism, evidence, safety, and access plus the upstream genetic variables that apply regardless of which compound a provider ultimately discusses

Furthermore, a comprehensive study utilizing Glp-1R Tcell+/+ and Glp-1R Tcell/ mice demonstrated that, in the setting of T cellmediated inflammation within IELs, the anti-inflammatory and tissue-protective effects of GLP-1RAs, both systemically and in the gut, were abolished in the absence of GLP-1R expression in T cells [165]